Chapel End Surgery – Dr Ganapathi – Library – Behind the Headlines

The 9-year-old, he married his wife of 62 years, for the second time – in 2013, 8-year-old schoolboy South Africa Saneie Masilela made headlines for marrying a 61-year-old woman South African. Vlachs (English pronunciation: /ˈvlɑːk/ or /ˈvlæk/) is a historical term used for Eastern Romance-speaking peoples in the Balkans and Eastern Europe; Exonym for several modern peoples from the population in present-day Romania and Moldova, the southern Balkan Peninsula and south and west of the Danube. Pantomime character puss in boots then subscribe to the learning and there is no conclusive etiquette on when. The Daily Telegraph says a vaccination programme could be in place by late 2010 if it is proven to be cost-effective. Vāmācāra (Sanskrit: वामाचार, Sanskrit pronunciation: [ʋɑːmɑːcɑːrə]) is a Sanskrit term meaning “left-handed attainment” and is synonymous with “Left-Hand Path” or “Left-path” (Sanskrit: Vāmamārga). Vadavannur is a village and gram panchayat in the Palakkad District of Kerala, India. Varacin is a bicyclic organosulfur compound originally found in marine Ascidiacea from the Polycitor genus.

The Vaganova Academy of Russian Ballet is a school of classical ballet in Saint Petersburg, Russia. Vāmācāra (Sanskrit: वामाचार, Sanskrit pronunciation: [ʋɑːmɑːcɑːrə]) is a Sanskrit term meaning “left-handed attainment” and is synonymous with “Left-Hand Path” or “Left-path” (Sanskrit: Vāmamārga). Notable people with the surname include: The Vlachs (endonym: Rumâni, Serbian: Власи / Vlasi) are an ethnic minority in eastern Serbia, culturally and linguistically related to Romanians. When there is a damaged backlink we’re not in control of it. Shingles, or herpes zoster, is a painful skin rash that is caused by a reactivation of the varicella zoster virus (VZV) that causes chickenpox. Anyone who has had chickenpox could develop shingles, although it is more common in people over 60 years old. The initial symptoms of shingles include oversensitivity and a burning sensation in the affected area followed by the development of a rash of small raised spots that soon turn to fluid-filled blisters.

After about a week, the blisters dry to form scabs that eventually fall off. The rash, which usually only affects one side of the body, can be very painful and debilitating. (‘Multum’) is accurate, up-to-date, and complete, but no guarantee is made to that effect. It is widely believed that the dish was brought to the United Kingdom by returning British troupes from India. The lingering pain from shingles is known as postherpetic neuralgia. Chickenpox is an infectious disease usually affecting children, although it can be caught by any person of any age if they have never had the disease. It is extremely common in children aged between two and eight years.


Jane Seymour is not like the average woman 62 years old. Symptoms vary from child to child, but generally, they are mild and disappear in seven to ten days. Pure and boyish in her athletic but sometimes it a combination back to services protective. Following a chickenpox infection, a person’s body will make antibodies, making them immune to further new chickenpox infections for the rest of their life. However, the virus remains dormant in the nerve cells and can be reactivated years or decades after the primary infection. When it reactivates, it causes an outbreak of shingles, usually following the route of a nerve supply to the skin. The risk of shingles increases with age, and while it unclear what triggers a reactivation of infection, it is thought to be due to compromised immunity.

Possible reasons for a weakened immune system can be the use of certain medications, illness, malnutrition or because of the natural decline in immunity that can accompany increasing age. Another possible treatment is the use of antiviral medicines that, while unable to cure shingles, can limit the severity of infection by helping to limit the replication of viral particles in the body. Antivirals are most effective if taken within 72 hours of the rash appearing. Antiviral drugs are often prescribed to people aged 50 years or above, those that have a weakened immune system or people with severe infection. A person with shingles cannot spread shingles to other people. However, the varicella zoster virus is present in the fluid-filled blisters of a shingles rash, meaning exposure to people with shingles can lead to chickenpox in people that have not already caught the virus or been vaccinated against it. While chicken-pox is often a mild illness in childhood, in adults it can be more severe.

Certain groups, including pregnant women, are also at a heightened risk of complications from chickenpox infection. A person who has had chickenpox is at no risk of catching shingles from someone with shingles or chickenpox again because of the lifelong immunity provided by their own chickenpox infection. Almost everyone has oral herpes, including me · Newborn saves her  dating site hot or not Get information on dating with a sexually transmitted disease. It had special rights and privileges, including trade monopolies, remaining one of the most powerful force for over 250 years. The vaccine will be made of a weakened chickenpox virus that will stimulate an immune response to make the body immune to future infection. According to the Centers for Disease Control and Prevention, there has never been a case of transmission of chickenpox virus from a person who has received the shingles vaccine. However, some people who receive the vaccination may develop a “chickenpox-like rash” near the site of their injection, so as a precaution the area should be kept covered.

As the vaccine is a live vaccine it will not be given to people who have severely compromised immune systems (due to illness or immune suppressive medication) or to pregnant women. Research described by the US Food and Drug Administration (a study of 38,000 people) showed that vaccination reduced the occurrence of shingles by 50%. Play driving games at free online games than games min uploaded by wharfitmaria bamford ask me about my new god. The JCVI will release a full statement and recommendations to government about the scope of a universal vaccination programme in this country. They have advised that a programme be initiated if “a licensed vaccine is available at a cost effective price”. The statement also suggests that any such vaccination will only be available for people aged 70 to 79 years old. The Guardian has reported that some campaigners want the vaccine to be offered to people from the age of 60 years, when the risk of shingles rises sharply.

However, it is important to remember that the JCVI has considered a range of studies assessing the effectiveness and safety of the vaccine and have come to their judgement by following this systematic analysis of evidence. There may explanations justifying this decision in the full statement that is due to be released.

Herpes of the eye, or ocular herpes, is a viral-[Good Tcm Net]


foods. Ocular shingles is a condition that occurs when the herpes virus infects the tissue of the eye. The most common allergies are those related to pollen, particularly when the weather is warm and dry. Clinical trials involve medical research with people. Prompt treatment with anti-viral drugs helps to stop the herpes virus from multiplying and destroying epithelial cells. Epithelium The epithelium is the cornea’s outermost region, comprising about 10 percent of the tissue’s thickness. Yesterday I found out I have hsv-1 and 2.

What are the side effects of ointment aciclovir dosierung bei herpes thuoc clopidogrel 75mg podczas karmienia piersia herpes tratament. The part of the epithelium that serves as the foundation on which the epithelial cells anchor and organize themselves is called the basement membrane. You can add them to your ocular herpes eye drops bathwater or you can make use of Melissa. Also, the antioxidant formula in the study did not have any significant effect on the development or progression of cataracts. This type of visual impairment is called corneal blindness. Then there are the fake diseases conjured up by the nefarious multi-billion-dollar vaccine industry, and propagated by the CDC, and hyped to the fullest extent by the mass media, withlies about breakouts and pandemics, so that everyone will rush to the nearest doctor, pharmacy or Walmart and get stuck with a needle full of MSG, mercury, formaldehyde and aluminum, not to mention combinations of genetically engineered organisms, bacteria andlive viruses there really may not be a cure for. [Read more about uveitis.] When this infection occurs in the retina or the inside lining of the back of the eye, it is known as herpes retinitis.

Virus Evasion of MHC Class I Molecule Presentation

The first important clue to the regulation of HSV by chromatin was the observation that a virus encoding a mutant VP16 that lacked the protein’s activation domain exhibited accumulation of nucleosomes on the viral IE gene promoters [28]. let us help you regain your life back with our comprehensive natural treatment for herpes.. Herpes zoster | definition herpes zoster medical, Looking for online definition of herpes zoster in the medical dictionary? Genital herpes health center – webmd, Learn genital herpes (hsv-1, hsv-2), symptoms, signs, information treatment. join free for dating with herpes, hpv and hiv aids positive singles.. of the five modified rifles used on the show, one. $1.5 mil is pretty steep for genital herpes.

its purpose is to explain.Eurican herpes 205 vaccine – viovet, Eurican herpes 205 is specifically indicated during pregnancy. dermnet nz, Herpes zoster what is herpes zoster? – cervical cancer advice ., What genital warts ? Greater minneapolis council churches, Greater minneapolis council of churches 1001 e. In contrast, a similar sized improvement in performance at the end of training would require finding the platform in 100 cm, a result that would be most surprising given the established baseline performance in the water maze. Fibroproliferative diseases can cause dysfunction of every major organ system and as a class of disorders is a major cause of death in the United States. Peptides are provided by proteasomal cleavage of ubiquitinated cytosolic proteins and TAP transport into the ER (2 ), and both TAP and the proteasome are known targets for viral interference (17 ,18 ,19 ,20 ,21 ,22 ,23 ,24 ,25 ,26 ).

Within the ER, the peptides are N-terminally trimmed by ER aminopeptidase associated with Ag presentation (ERAAP) (3 ). The structure of Rep later confirmed a dimeric protein containing two subunits in different conformations.31 Unlike Rep, PcrA crystallizes as a monomer, but PcrA assumes different conformations in the presence and absence of ATP.30,32 Alterations between these conformations have been proposed to allow the monomeric PcrA helicase to crawl along DNA like an inchworm. This extra level of quality control performed by the PLC distinguishes MHC class I molecules from many other proteins that assemble within the ER. The spliceosome complex contains four small nuclear ribonucleoprotein (snRNP) particles (U1, U2, U5 and U4/U6), each of which contains the corresponding snRNAs and a set of specific and common proteins (reviewed by Krämer, 1995, 1996; Will and Lührmann, 1997). these treatments for herpes. We demonstrate that injury-induced neurogenesis occurred throughout the dentate gyrus and these neurons stably incorporated themselves within the hippcocampus and elaborated sophisticated dendritic arborizations. Some virus proteins facilitate the endocytosis of MHC class I molecules by ubiquitination (33 ,34 ,35 ,36 ,37 ), and one of these proteins, the Kaposi’s sarcoma-associated herpesvirus K3 protein, has been specifically shown to direct these endocytosed MHC class I molecules to the lysosome by tumor susceptibility gene 101-dependent sorting (38 ,39 ).

3(1):5-11. The strongest evidence is for roles for Nef and Tat in this process. Writing on infections of humankind, Tony McMichael and I [5] have called those that cospeciated with their hosts ‘family heirlooms’ and those that crossed over from other hosts in recent evolutionary time ‘new acquisitions’. Nef increases the pathogenicity of HIV in several ways, including down-modulation of cell surface MHC class I molecules (28, 30). To date, DAI expression has not been reported in microglia or astrocytes, and a role for this putative viral sensor in the innate immune responses of resident CNS cells to viral challenge has not been explored. Although the cause is unclear, searching for an antecedent exposure history prior to lymphoid malignancies is helpful in determining the causes of the geographical differences. Look, if there’s a tactical or strategic reason to walk him, then go ahead.

The ability of US3 to modulate cofilin activity levels is underscored by the fact that transfection of a WT US3-encoding construct in ST cells was sufficient to suppress phospho-S3 cofilin levels, as shown in Fig. The mechanism of MHC down-modulation may be MHC allele dependent and/or cell dependent (10). For example, the degree of MHC class I cell surface reduction by Nef varies ∼100-fold depending on the type of cell examined (28, 30, 42, 44), suggesting the involvement of cell-specific factors that either assist or interfere with Nef’s activity. Figure 2. For example, HIV-1 down-regulates HLA-A and -B but has little impact on the expression of HLA-C and -E; this selectivity allows HIV-1-infected cells to escape lysis by NK cells (45). Physical interaction has been demonstrated between Nef and particular amino acid residues present in the cytoplasmic tail of HLA-A2, but not in HLA-E and in site-directed HLA-A2 mutants (27). Importantly, the identification of specific binding sites for Nef on MHC molecules may lead to an understanding of differences in AIDS susceptibility or resistance that are linked to particular MHC alleles.

In contrast to its effect on HLA-A2, Nef has very little effect on murine MHC class I molecules (46), a finding that further adds to our appreciation of the difficulty of deriving a suitable small-animal model for AIDS. Studies with mouse/human MHC chimeras indicate that amino acid residues in the extracellular domains of the MHC molecule, as well as in the cytoplasmic domain, can play a role in Nef-mediated MHC class I down-modulation (46). The HIV-1 tat gene encodes a protein that transactivates HIV transcription (47). Tat is able to repress MHC class I promoter activity, as well as the activity of the promoter for β2m (48, 49, 50, 51). Tat is also capable of inhibiting the association of the 11S regulator subunit with the proteasome via a shared binding site, interfering with the production of peptides for MHC binding (17, 18). Tat is also secreted by HIV-infected cells (52). The presence of extracellular HIV-1 Tat indirectly affects MHC class I presentation by inhibiting dendritic cell phagocytosis of apoptosed cells (53, 54).

However, Tat’s effects on dendritic cells are complex. In vivo expression of the type I collagen-HSV-TK transgene. Once internalized, Tat induces dendritic cell maturation and thereby, in an interesting twist to the Tat story, causes up-regulation of cell surface MHC class I (as well as MHC class II and costimulatory molecules) (55). In addition to the effects of Nef and Tat, there are also hints that HIV-1 Vpu may increase the turnover rate of nascent MHC class I H chains (14), and that an as-yet-unidentified HIV-1 protein may block TAP transport of peptides into the ER (21). Although no F4 helicases from pathogenic human viruses have been reported, an SF3 helicase from human papillomavirus has been extensively studied both mechanistically and structurally.48 This protein could be an important target for treatments of this class of tumor viruses. Apart from the sequence polymorphism, MHC class I molecules have also evolved alternative pathways for accessing pathogen-derived peptides and potentially overcoming the plethora of inhibitory mediators. Overlays of images obtained from the antibody-stained and the YFP–HCF-1-transfected cells show that HCF-1 co-localizes with splicing snRNPs, gems and Cajal bodies in HeLa cell nuclei (Figure G, K and O).

Notably, such an observation underscores the importance of not restricting studies of MHC/virus interaction to the examination of the effect of a single virus protein in isolation. Cells in the counting frame (40 μm X 40 μm) were counted under a 40X objective lens. The mechanism for this effect has been demonstrated to be ubiquitination of the MHC class I H chain (or a protein associated with it), and the effect can be blocked by protease inhibition (35, 36). Gekker G, Hu S, Sheng WS, Rock RB, Lokensgard JR, Peterson PK. A related virus that is a horse pathogen, equine herpesvirus-1, has likewise been shown to cause endocytosis of cell surface MHC class I molecules (58). erectus. The mK3 protein has been demonstrated to assist the virus in escape from T cells during the latent phase of infection (59).


Glial cells were then collected from the interface and washed with PBS. MHC class I molecules in assembly complexes awaiting peptides are preferentially associated with mK3 (12), and, in fact, the assembly complex proteins TAP and tapasin are absolutely required for mK3’s effect on MHC class I (60). Not in the NBA there’s not. An ER-resident protein that can cause down-regulation of surface MHC class I is also expressed by myxoma virus, which is a poxvirus rather than a herpesvirus (61). A unifying characteristic of this myxoma protein, the Kaposi’s sarcoma-associated herpesvirus K3 and K5 proteins, and the murine γ-herpesvirus 68 mK3 proteins is that they all possess a particular type of conserved structural motif that is correlated with ubiquitin ligase activity. Indeed, the myxomavirus M153R protein has been shown to have ubiquitin ligase activity in vitro, and MHC class I cytoplasmic tail lysines are necessary for down-regulation of the MHC class I molecules by M153R (39). The second complex, ATAC, contains the histone acetyltransferases GCN5/ATAC2 and appears to target histone H3/H4 for acetylation [69,70,71,72,73].

Murine CMV (MCMV) expresses three genes that encode protein for blocking Ag presentation: m04, m06, and m152. By use of a set of mutant MCMVs with deletions of the three genes in all possible permutations, the effect on the surface expression of multiple MHC class I allele products was examined (62). These experiments showed that these MCMV proteins can have synergistic effects, but certain combinations of these proteins are actually antagonistic; thus, this approach revealed a truer picture of MCMV’s effects on MHC class I in the course of natural infection than had been hitherto obtained. Although there is substantial evidence for an MCMV gene, m152, affecting the presentation of a particular MCMV epitope in vitro, the immunodominance of the same epitope in vivo is not affected by whether m152 is expressed (63). Thus, the effect of MCMV proteins on cells that endogenously express them and their effect on professional APCs that take them up and present their peptides by cross-priming appear to be distinct. The impact of MCMV infection on APCs has also been examined in other recent studies. The results from these studies indicate that MCMV does induce down-regulation of MHC molecules on dendritic cells and macrophages; however, there are differences in the effects of immune evasion genes when APCs and fibroblasts are compared (64, 65, 66).

For example, expression of the m04, m06, and m152 genes are all necessary to block recognition of macrophages by Kb-restricted CTLs, and the m04 gene, in particular, plays a greater role in inhibition of T cell recognition in macrophages than in fibroblasts (66). Human CMV (HCMV) invests heavily in products able to interfere with MHC class I. The HCMV unique short (US) genes (US2, US3, US6, and US11) all assist HCMV in evading MHC class I presentation. HCMV encodes a unique long (UL) region protein, UL18, which is an MHC class I homolog, capable of binding β2m and peptide (67, 68, 69). The procedures for cultured cells were the same as that for tissue except that the cells were fixed directly with 100% cold methanol for 10 minutes. HCMV US2 (and US11) cause ejection of MHC class I H chains into the cytoplasm, which results in their proteasomal degradation (15, 16). The US2 cytoplasmic domain has been shown to have major involvement in this process (71, 72).

Although studies in cultured cell lines did not distinguish US2 and US11 in effectiveness, new experiments with primary human dendritic cells have revealed that US11 is much more effective than US2 at degrading MHC class I in these cells (73). US3 possesses a novel, noncontiguous ER retention sequence, and binds to MHC class I to prevent its egress to the cell surface (4, 5, 74). These results indicate that HCF-1 stably associates with the human spliceosome complex and may function in the pre-mRNA splicing pathway. Inhibition of TAP has been used successfully as a defensive mechanism by other herpesviruses, including a swine pathogen, pseudorabies (25), and bovine herpesvirus 1 (26, 76). The Morris water maze task was conducted similar to the method previously described (Powell et al., 2004; Tabuchi et al., 2007; Etherton et al., 2009). US8 does not quantitatively affect MHC class I surface expression (77), although its influence on MHC class I-restricted Ag presentation remains to be more fully explored. 175(7):4189-93.

The adenoviral protein E3/19K was first shown to down-regulate cell surface MHC class I expression by direct retention of MHC H chains in the ER (7, 8). Rantala [20] suggested that nakedness could have had a selective advantage to rid the body of lice and other ectoparasites, a view also championed by Pagel and Bodmer [21], who added that being seen to be free of lice would be a fitness indicator and a good mating strategy. As a result of binding to TAP, E3/19K blocks the association of TAP with tapasin (19). To determine if glial cells express DAI in situ, we first assessed the expression of this viral sensor in ex vivo microglia/macrophages and astrocytes isolated from uninfected mouse brain tissue. APLP2 is a type I transmembrane protein and a member of the amyloid precursor protein family (78). Because she’s a skank. Thus, by facilitating MHC binding to APLP2, E3/19K can act via APLP2 to inhibit MHC surface expression.

In total, a single adenovirus protein, E3/19K, can attack MHC class I by three separate mechanisms, serving as a highly resourceful viral weapon against the cellular immune response. Cellular transformation by adenovirus type 12 (Ad12) induces an oncogenic phenotype (80). It is now clear that epigenetic regulation of HSV infection is an important contributor to the determination of the outcome of infection and represents a supra-regulatory overlay beyond the contributions of individual DNA binding transcription factors [98,99,100,101]. Relevant to surface down-regulation of MHC class I, Ad12-transformed cells have decreased expression of the MHC H chain, TAP, tapasin, LMP-2, LMP-7, MECL-1, and PA28 (20, 82, 83). Reduced expression of these proteins has been attributed to an IFN-related effect (84, 85), and, in the case of MHC class I transcriptional down-regulation, dual mechanisms (i.e., repressive COUP-TFII and inactivated NF-κB) are used, evidently as mutual back-up measures (86). Two reports in the last year have indicated that the bovine papillomavirus E5 protein can down-regulate the surface expression of MHC class I molecules (87, 88). Cells that express E5 have reduced levels of MHC class I mRNA and protein and surface MHC class I, whereas surface expression of a control protein, the transferrin receptor, is not affected (87).

E5 can retain MHC class I in multiple cell types (87), and the site of blockade has been shown to be the Golgi (88). Because E5 is a viral oncoprotein, this finding is of interest from the perspective of tumor, as well as virus, escape from the specific immune response. An important new trend in the analysis of MHC class I/virus interactions, evidenced by several of the studies described above, is the rising frequency of productive investigation of these interactions in the context of virus infection, using gene-deletion virus variants and samples from infected patients. The results obtained with these approaches are providing us an appreciation of complementary and antagonistic interactions among the proteins expressed by each virus. Certainly, a future generation of these studies will involve analysis of the impact on MHC class I Ag presentation of infections with multiple different chronic viruses, as is often the case in the human situation. The effects of viruses on MHC class I function are also being analyzed in recent studies in a wider variety of cell types than ever before, including professional APCs. In addition to HIV and MCMV, other viruses, e.g., human herpesvirus 3 (also known as varicella-zoster virus), infect dendritic cells and down-regulate expression of MHC class I molecules on them (89).

Cells were isolated from sponges 21 days after sponge implantation. Although the selective pressure on viruses to down-regulate MHC class I expression for the purpose of avoiding CTL killing is apparent, the ability of viruses to escape from CTLs without simultaneously increasing susceptibility to NK cells is intriguing. Several recent studies have dealt with this issue, and in the process have generated a new immunology subfield: virus interference with NK cell recognition. In one case, the host has turned the virus’s weapon on itself. In a story replete with evolutionary twists, a cell surface MCMV protein encoded by m157 binds to an inhibitory NK cell receptor that is expressed by an MCMV-susceptible mouse strain, and the same m157 product binds to an activating NK cell receptor in MCMV-resistant mice (90). Taken together, these results suggest a possible role for HCF-1 in either splicing catalysis or a late stage of spliceosome assembly. For example, the m152 gene product of MCMV, which inhibits the MHC class I expression, also affects NK cell reactivity against MCMV by modulation of NKG2D ligands, specifically retinoic acid early inducible 1 proteins (11, 91, 92, 93).

Further analyses were conducted using post-hoc Tukey HSD tests and planned comparisons (contrast analysis and additional ANOVAs). In other cases, viral proteins that block NK activity interact with MHC-like molecules. 47(4):358-66. Thus, the viral defense strategies against CTLs and NK cells are intertwined, in a fabric that is fascinating to unravel. It is noteworthy that the prevalence of infestation by pubic lice seems to be decreasing among women and men who remove their pubic hair using ‘bikini wax’, rather as the Renaissance painters discreetly depilated classical female nudes. These viral defense mechanisms are not without price, because it has also been discovered that expression of immune evasion proteins can permit the host to present and recognize peptides from those same proteins (96, 97). As shown in Figure , HSV-1 induced the production of soluble mediators by microglia that decreased CATH.a neuron-like cell viability as assessed by changes in cell attachment and trypan blue exclusion in an MOI and time dependent manner.

↵1 This work was supported by National Institutes of Health Grant GM57428 (to J.C.S.), by National Institutes of Health Training Grant T32 CA09476, and by National Institutes of Health Individual National Research Service Award Fellowships (to J.L.P. and C.R.M.). ↵3 Abbreviations used in this paper: β2m, β2-microglobulin; ER, endoplasmic reticulum; TAP, transporter associated with Ag processing; MCMV, murine CMV; HCMV, human CMV; US, unique short; UL, unique long; APLP2, amyloid precursor-like protein 2; Ad12, adenovirus type 12.

could i contract herpes from my boyfriend?


A California student-athlete says his wrestling career is over after he contracted a highly contagious form of herpes during a match. State interscholastic officials said they won’t cancel the tournament in Bakersfield, insisting they follow rigid protocols to protect wrestlers from infections and viruses like “herpes gladiatorum,” which is spread mostly through red skin lesions. I have a girlfriend and we were wondering what the chances of me spreading it to her by kissing are. “I’ve gotten a lot of supportive comments — 100 percent,” Blake said Thursday, just two days after he was diagnosed with the “herpes gladiatorum” virus that will go dormant, but can flare up for the rest of his life. What’s the history of this test in terms of accuracy, and are there new tests that are actually accurate? Not his genitals. He says he helped institute at the school some of the strictest hygiene standards in the sport, including requiring wrestlers to step in a pan of disinfectant before they step on a mat.

Herpes gladiatorum, or mat herpes, is usually caused by the virus hsv-1. IBM’s Watson is a computer system that could answer questions asked in natural language. Your boyfriend probably caught it when the mouth of someone infected with hsv-1 orally came into contact with a cut on his forehead. Herpes can’t normally enter through normal skin, it is too thick, it has to enter through a cut or abrasion. We scored twice and if we had just a little more luck and we would have won the match.

, HSV-1 Is Increasingly Identified As The Cause Of Genital Herpes In Australians

The shingles vaccine is very safe and effective at preventing shingles. Professor Frazer, a former Australian of the Year, developed the experimental vaccine with his team at Coridon, a biotechnology company he founded in 2000. (ASX: AHZ) has reported successful results of an initial study of a novel Human Papilloma Virus (HPV) vaccine, being developed by its investment company Coridon, that shows protection against tumour development in mice. All participants were screened to exclude previous exposure to Herpes simplex virus 1 (cold sores) or HSV-2 (genital herpes. Proactive and Baillieu Holst have really understood this AHZ stock more than any others, Proactive seem to know more about Pr Frasers future plans than any of us, hints from them all the time and Baillieu’s research and detail is second to none, yes i say reputable brokers in my book, oh yeah throw in morgans, pattersons, motley fool and others who are no more or less reputable than this folk. A vaccine to treat herpes simplex has been found safe to use in humans, further trials will test its effectiveness. Reasons for the seemingly increasing prevalence of anogenital HSV-1 are unclear.

Safety of Herpes Zoster Vaccine in the Shingles Prevention Study: A Randomized Trial. Et al Increase in rates of herpes simplex virus type 1 as a cause of anogenital herpes in western Sydney, Australia, between 1979 and 2003. The Centers for Disease Control and Prevention in the U.S. We would note that the company’s shares were priced at $0.026 a year ago (compared to the current price of $0.105) and that our previous guidance of $0.07 made in June 2013 – when prices ranged from $0.04 to $0.059 – was surpassed in just two month on 19 August. Recently, the number of reported genital herpes cases caused by type 1 virus has increased. Herpes is very infectious, it’s one of these viruses you can catch from a single contact from someone who’s got it, Professor Frazer said. In recent years, an increasing proportion of genital herpes cases have been found to be caused by the type 1 virus (6, 8, 9, 11), particularly in the United Kingdom (14, 15).

In the key pre-licensure clinical trial (N Eng J Med 2005;352:2271-84), vaccine recipients had a 51 reduction in shingles and less severe illness when shingles did occur compared with placebo recipients. HSV Type 2 is the commonest cause of genital herpes and is almost always sexually transmitted. Infection with genital herpes from HSV Type 1 is increasing worldwide due to oral/genital tranmission of the virus. After Human Papilloma Virus was detected in cervical cancer tissue, it was hypothesis ed that HSV 2 might initiate mutations and carcinogenesis in HPV infected cells. Herpes simplex virus is also responsible for cold sores that occur on the. Professor responsible for cervical cancer vaccine successfully trials new treatment. While both HSV-1 and HSV-2 can cause genital herpes, HSV-2 is the primary cause.

Immunization enables the body to better defend itself against diseases caused by certain bacteria or viruses. Herpes simplex is a viral disease caused by Herpes simplex viruses; both herpes simplex virus 1 (HSV-1) and herpes simplex virus 2 (HSV-2) cause herpes simplex. U.S., HSV-1 is increasingly identified as the cause of genital herpes in Australians; Causes. While genital herpes is considered to be a sexually transmitted infection, any form of contact is a possible route of infection. However, infection may occur at virtually any area of skin, with increasing risks when skin integrity is compromised. HSV-2 genital herpes affects up to one in eight Australians, and there is currently no curative treatment, Professor Frazer said. Type-specific seroprevalence data may be useful to identify particular population subgroups that have a notably higher risk of HSV infection 8.

Most vaccines are safe and cause few if any serious side effects. HSV-2 prevalence varies between countries, being higher in the USA than in Europe, Australia and New Zealand. Age: The incidence of new infections is highest amongst young adults, but as infection is lifelong, overall prevalence increases with increasing age. Furthermore, HSV-1 has become an important cause of genital herpes in some developed countries. Seroprevalence of HSV-1 decreased but the incidence of genital herpes caused by HSV-1 may be increasing. The trial is designed to demonstrate the vaccine’s safety and tolerability, as well as determining the effective dose and that it generates a robust immune response. Cold sores are caused by herpes simplex virus (HSV).

Who is at risk for shingles? Although rare in Australia, there is a risk that you can pass HSV infection to your baby in pregnancy, during the birth or immediately after birth. Neonatal Herpes Simplex Virus (HSV) infection is a rare but serious condition that can present with sores and lesions to the skin, eyes, and/or mouth, encephalitis, or disseminated infection affecting multiple glands and organs. However, more recent research shows an increase in genital herpes caused by HSV-1 in Australia, the United States, and elsewhere. The reasons for this increase are unknown but preventive efforts should include increasing adolescent awareness of sexually transmitted infections like HSV. Genital herpes is an incurable sexually transmitted infection. Most cases of genital herpes in humans are caused by the herpes simplex virus 2 (HSV-2), which affects around 12 per cent of Australian adults.

How can I learn about or participate in clinical research? Both herpes simplex viruses 1 and 2 produce similar symptoms, but they can differ in severity depending on the site of infection. Genital herpes, which typically affects the penis, vulva, and rectum, is usually caused by HSV-2, although the rate of HSV-1 genital infection is increasing. Genital herpes simplex is caused by infection with the herpes simplex virus (HSV). Type 1 is the usual cause of infections of the oral region and causes cold sores (herpes labialis). Can you pass? DNA detection by PCR is increasingly the more common method used.

This is Why Baking Soda is One of The Most Powerful Things You Can Use. Here’s What You Didn’t Know About It –

Pimples on your vagina may be caused by skin conditions other than acne from clogged pores. Baking soda or sodium bicarbonate is extremely cheap, but a very powerful ingredient. Plus, it’s extremely beneficial for oral health, personal hygiene, skin and hair care and last, but not least, as a cleaning agent. If you want after hard work out to relieve muscle pain you should drink solution of baking soda. As we already mentioned you can use baking soda for a long list of things but do you know how? Gargle with baking soda and water to freshen your breath. You need to mix 1tbs.

Use it to eliminate plague and colds. Mix 3 tablespoons of baking soda, 1 teaspoon of salt, peppermint essential oil and a small amount of water. Mix 3 tbs. You can also use baking soda to refresh your breath – just gargle a little bit of baking soda. water and rub gently on your face. If you suffer from oral thrush then gargle a mixture of baking soda and water; Treat UTI with a mixture of 230 ml of water and ½ tsp of baking soda. baking soda, 1 tbs.

Antivirals – OTC (over-the-counter) cold sore creams. They are going to dry much faster. –  Sprinkle baking soda on scrub nail and damp nail brush to eliminate grime and remove nail fungus. Combine 1 teaspoon of baking soda, with 1 teaspoon of water and then rub your face. Don’t use it while you’re on a low-sodium diet, as it can deplete the body of essential nutrients. — Drink a mixture of baking soda and water to relieve muscle pain after strenuous exercise. She did a strep test on me just to make sure I wasn’t carrying any contagion.


— Mix 1/2 tsp. If you want to reduce acid reflux you should make alkaline environment in your organism. water and drink to relieve heartburn, acid reflux and GERD symptoms. — Mix 1/4 – 1/2 tsp. of water and drink the mixture. Carry on this treatment after every 3 hours throughout the day. It will foam and fizz, so use a tall glass and wait for all bubbling to stop.

Then add 8 oz. It will neutralize your pH, protect stomach acid and decrease acidosis. Wash it off and pat dry the infected area. — Douche with a mixture of 1 tbs. Or, you can also treat UTI by preparing a drink with 230 ml of water and ½ teaspoon of baking soda. Also, do not use baking soda home remedies for more than a two week period without consulting your doctor. — Treat a urinary tract infection by drinking an 8 oz.

glass of water mixed with 1/2 tsp.baking soda once or twice a day. — Relieve a thrombosis by drinking the mixture of baking soda and lemon juice or apple cider vinegar mentioned above. Here’s what you need to do – you just have to combine lemon juice or apple cider vinegar with baking soda. Here’s what you need to do – you just have to make a mixture of warm water and a teaspoon of baking soda and use it. Mark Sircus discusses the findings of Dr. Tullio Simoncini with regards to cancer being a yeast overgrowth and explores treatment protocols with baking soda. And never use baking soda remedies longer than 14 days in a row without having consulted your GP.

It should not be used on a low sodium diet unless under supervision. In some instances, it may interact with prescription drugs, so check first with your pharmacist. Only use baking soda after it’s been dissolved in liquid and never consume it in dry powder form. Do not use baking soda home remedies for more than a two week period without checking with your healthcare practitioner. Baking soda should not be used by pregnant or nursing women or by children under the age of 5. Thanks for taking the time to read this article. If you found this information helpful, please share it with your friends and family.

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After a lackluster episode, Brooklyn Nine-Nine felt like it bounced back for “Tactical Village.” This was a more mature episode, both structurally and in the way it pushed the relationships of each of its characters forward. That’s because we’re changing things up this year to better suit the sketch format. However, I didn’t climax during sex, which is a result of years of death-grip masturbation. Embarrassing, you may think, but surely tolerable. On Tuesday night’s “Savage U” season finale, Savage is headed to Texas Tech, and the residents of Lubbock may be in for quite a shock when they hear what sexual euphemism the sex columnist bestowed upon their quaint Texas town. It’s driving me nuts because I feel like I’m missing out on a big part of my life. While I’m prepared to sign off on women in thong underwear and while I recognize that male strippers and gay porn stars must wear thongs to work, I refuse to sign off on straight men in thong underwear.

However, condoms do not provide complete protection, because a herpes lesion may not be covered by the condom and viral shedding may occur. A flasher may be the first thing that pops to mind when people hear the term “exhibitionist,” but while all flashers are exhibitionists, it doesn’t follow that all exhibitionists are flashers. “That will help to separate the fantasy of the humiliation from the reality of their strong relationship. Still, Guy is nice, so Girl keeps seeing Guy. Any advice on approaching a “hey bro, I want to renegotiate rules” conversation? The only way to get the edgy sex life you want, SKARE, is to accept that edgy sex lives always involve a certain degree of risk. Hell, there’s no such thing as risk-free anything.

What if the reality is shattering? But some risk will creep into your condom-free sex after you start having sex with other people, WTR—even if you’re using condoms. Your risk of getting an STI will be much, much lower if you use condoms—consistently and correctly—with those other partners, but sex with other partners will introduce some risk. But those who post misleading photos have no one to blame for their hurt feelings but themselves. No, he could have bad luck or one terribly reckless moment or wind up in a relationship with a guy who lies to him. Late in the movie, Carmen is stabbed through the chest by an enormous and very sharp bug leg, pinning her to the ground.

GENITAL HERPES BASIC INFORMATION – Viral Infections Blog Articles

There are over 20 sexually transmissible diseases. In other words, a individual may be contaminated for years before that individual knows that the disease is there. Genital herpes virus can go forgotten even with the infected mortal himself. HSV-1 usually causes lip sores (fever blisters, cold sores), but it can cause genital infections, too. Practice good genital hygiene. HSV is transmitted only while lesions are present. Assumes particles of viral.

Genital herpes itself is not deadly, however it can increase your susceptibility to life-threatening infections such as HIV. This is because the newborn is exposed to the mother’s blood and other bodily fluids during birth. This infection, known as congenital herpes, can be fatal in newborns. Herpes simplex virus type 2 (HSV-2) is the leading cause of genital ulcer disease worldwide. Have we learned nothing about the practices of safe sex and protecting ourselves and each other since 1907? More blacks than whites have herpes. The group in which herpes is proliferating most quickly is young white teens; in those who are age 12 to 19, HSV-2 was five times more prevalent at the start of the new millennium than it was two decades earlier.


All the adverse reactions have been outweighed by its positive results provided it will be used effectively. An outbreak can occur anytime that’s why even without symptom, having unprotected sex with an infected mortal is still like playing with fire. Lacking any awareness that they have genital herpes, these individuals often spread it unknowingly. This obviously poses an enormous health risk for those who are sexually active and underscores the importance of STD testing before initiation of a sexual relationship with a partner. This disease has major health consequences because the virus stays in the body in certain nerve cells, periodically causing lifelong symptoms in some but not all individuals. Stress, illness, poor nutrition, excessive activity, and sunlight have all been known to trigger bouts of herpes in herpes sufferers, even when the disease has lain dormant for a long time. Types of Herpes Genital Herpes – Genital herpes is typically caused by exposure to HSV-2, which is highly contagious and can be transmitted through direct contact with an infected individual, both sexual and platonic.

This is an infection of the fallopian tubes, uterus, and cervix. Flulike symptoms, including fever and swollen glands, are not unusual. Maternal outcomes at 2 years after planned cesarean section versus planned vaginal birth for breech presentation at term: the international randomized Term Breech Trial. Therefore, always better to use caution whether you’re experiencing symptoms or not. The site of the infection hosts the first sores (lesions), but these also can occur inside the vagina and on the cervix in women or in the urinary passage of either sex. Small red bumps morph into blisters, finally turning into painful open sores. This is another step towards healing.

Some people with genital herpes experience headache, fever, muscle aches, painful urination, vaginal discharge, and swollen glands in the groin. The primary episode of genital herpes is usually the worst and is often followed by four to five more symptomatic periods the first year. However, many who have HSV-2 experience no symptoms, and in some people, the symptoms are mild, but this disease can also cause painful genital ulcers that recur frequently. What sometimes makes herpes hard to detect is that it manifests itself in different forms. Some are easily missed; others are overt and dramatic. Detoxification – A tailored detoxification plan will follow the patient’s diagnosis. For males, the healthcare provider inserts a thin swab into the tip of the penis to retrieve a sample of fluid from the urethra.

It also can infect the urethra and cause burning. Against genital herpes (HSV-2), estimates of efficacy range considerably; A low incidence of breakage was reported for both condom types during appropriate use. Genital to genital, mouth to genital, mouth to mouth (if the infection is already on the mouth) etc.Some are also spread through the birth canal from mom to baby. Small sores in the urethra can cause painful urination. Aching or itching during the menstrual period is another symptom. Some women mistakenly think they are having a skin irritation caused by sexual activity when it is actually caused by herpes. Men who contract herpes may initially believe that they have acne, irritation caused by sexual activity, or jock itch.

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Type B-HBsAg positive; acute or chronic S See specific recommendations for care of patients in hemodialysis centers 778 Type C and other unspecified non-A, non-B S See specific recommendations for care of patients in hemodialysis centers 778 Type D (seen only with hepatitis B) S Type E S Use Contact Precautions for diapered or incontinent individuals for the duration of illness 1068 Type G S Herpangina (see enteroviral infection) Hookworm S Herpes simplex ( Herpesvirus hominis ) Encephalitis S Mucocutaneous, disseminated or primary, severe C Until lesions dry and crusted Mucocutaneous, recurrent (skin, oral, genital) S Neonatal C Until lesions dry and crusted Also, for asymptomatic, exposed infants delivered vaginally or by C- section and if mother has active infection and membranes have been ruptured for more than 4 to 6 hrs until infant surface cultures obtained at 24-36 hrs. Imlygic is a type of immunotherapy that uses a specially designed virus to destroy cancer cells. The virus is a genetically modified version of the herpes simplex virus type 1. These risks are associated with the intensity and duration of immunosuppression. Any pregnant women with a history of, or signs or symptoms suggestive of, herpes lesions around the birth canal, must inform their obstetricians of this fact. The goal of cryotherapy is to freeze and destroy targeted skin growths while preserving the surrounding skin from injury. Potted plants should be avoided inside the home as well as the consumption of nuts, as they may be contaminated by fungi and fungal spores.

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Are we done with this idiot yet?

Early Life Michael Dwayne Vick was born on June 26, 1980, to a mother who was 16, and a father who was a year older. However the nickname “Ookie” is mostly used in the pacific northwest region. In her lawsuit, which does not specify monetary damages, Elliott states that she met Vick at a Virginia Beach nightclub in May 2001 and, shortly thereafter, began a close personal relationship with the football star (though the couple did not have sex until late-2002). The Daily News points out you CAN’T buy a “Ron Mexico” dog jersey. After wading through all the stuff about the Yankees and Red Sox I was able to find an on camera statement by Vick. Mark Felt, associate director of the FBI, leaked information about the Watergate break-in to reporters Bob Woodward and Carl Bernstein of the Washington Post, and kept his identity secret for more than 30 years, until 2005. According to Elliot, when she confronted Vick about her herpes diagnoses, he admitted that he had the disease and knew about it prior to their encounter.

BABAYYYY. Simpson, who, I am almost certain, will be given a wonderful position with the Philadelphia Eagles, once he has paid his debt to society and gets sprung from that Nevada prison. He saw the clerk eating a burger and the bag had Ronald McDonald on the side of it. This guy is trash. Posted by U of Md Terps–Let’s go Maryland (Member # 706) on May 22, 2007, 05:51 AM: Yet Clinton Portis says if he is fighting dogs on his own property, that’s his business only. I suppose that goes for boning underage girls as well. Nike, don’t let me down.

Last night at the pub we got so tired of ESPN rooting for the Yankees we had to turn the sound off and turn on the jukebox. Posted by Another side (Member # 1698) on July 18, 2007, 02:04 AM: If the allegations are true, particularly the ones having him killing his own dogs, then Vick should go to prison. Ron Mexico[edit] That was the alias that Michael Vick used when he went to the doctors to get tested for herpes. Posted by writer2 (Member # 5966) on July 18, 2007, 05:25 AM: Just FYI–Vick is a huge story in Atlanta this morning. The young couple had four children but didn’t marry until just before the birth of the youngest child. Posted by Another side (Member # 1698) on July 18, 2007, 05:43 AM: I can. The Vicks are nothing more than common street thugs, hustlers.

That’s all they’ve ever been. Maybe one of those bobble head dogs people have in their car rear window. Probably so, but we had a basketball player at Okla. State named Richard Dumas who could have been great. But, Noooooooo….like Ricky Williams, he didn’t want to stop using drugs in exchange for the opportunity to be a millionaire for the rest of his life. Sheesh….. The judge was reportedly so infuriated with Vick’s bankruptcy plan that he ordered him back to prison.

The guy has a cannon for an arm…the wheels of a DB but a brain smaller than the one found in those dogs he was drowning! Posted by Great Cornholio (Member # 8365) on July 18, 2007, 07:05 AM: The thing that I find interesting is how the pundits are saying that Vick is only a “first time offender” in this new NFL arrest/crime policy. I understand that if this is a DUI/DWI case where it’s an isolated one night of his life situation, but according to the affidavit, Vick’s been doing this for 6 YEARS! answer. Phillips survived and refused to name the shooter so nobody was charged, it was reported, but it’s not an episode you’d think would reflect well on Vick. No Lakers, no Kobe, no Kevin Garnett and probably no LeBron James — plus Shaq may be hobbled. But “first-time offender” is a judicial term.

It means he faces more limited punishment, if found guilty, than if he had previous convictions. And the commish will go PacMan Jones on Vick. PacMan hasn’t been convicted has he? A federal indictment may be all the commish think he needs to shut Vick down until this is resolved. The Falcons and NFL will not “circle the wagons” around Vick until this is resolved legally. err fur off. They will break him off just like Nike.

Last season, Vick was fined $10,000 by the NFL and agreed to donate an additional $10,000 to charity after making an obscene gesture toward fans at the Georgia Dome. I was on the sidelines of a VT game last year. I thought I was caught in the middle of a either a Hell’s Angels rally or a meeting of the Crips. Originally posted by Ralphie the buffalo: The Falcons and NFL will not “circle the wagons” around Vick until this is resolved legally. Oi, just think of all the wondrous Old World traditions and mores one must give up to fit into the United States of America! They will break him off just like Nike. They may call it something else, but the effect will be the same.

I disagree, although I wish I didn’t. Arthur Blank has his proboscis implanted permanently up Vick’s tuckus. Even after sitting today and combing through the indictment papers – it’s too lengthy and detailed to be meritless – I will be shocked if the Falcons in any way suspend or punish Vick. The Falcons have been his enablers the past 7 seasons. I don’t expect that to change. Posted by Another side (Member # 1698) on July 18, 2007, 04:30 PM: I agree … vick thinks the panthers’ jerseys are hot (and i think he’d look hot in one), and with that as his main reason, has stated his want to play for carolina.


The bigger problem, I think, is they have no other QB, and do Atlanta fans want him playing? I think most people could probably get past the dog-fighting accusatons long enough to let hm take the field on Sundays, but the abuse and killing of his own dogs might be altogether different. It’s hard to forgive someone for shamelessly klling, in such barbaric fashion, their own animals. For those of you that aren’t familiar with Ron Mexico it’s an alias created by NFL Superstar Michael Vick of the Atlanta Falcons. Michael Vick is currently under investigation for hosting dog fights in his Viginia home. This story is not going to die, it’s only going to escalate as the NFL season approaches. BREAKING NEWS – Released on Tuesday, July 17th.

Vick Indicted! The death of Togepi due to a fractured skull. Posted by Jane Craig (Member # 4873) on July 19, 2007, 07:26 AM: I live in Atlanta and agree with Another Side. I do NOT want that man as our QB. He’s always been one reason why I haven’t glommed onto the Falcons. So forgive me for lacking passion about the guilt, innocence and/or punishment of one-time franchise quarterback Michael Vick for his alleged involvement in a dogfighting ring. Hell — given that the state, if inclined, can make a blind witness’ vision 20/20 — I’m even willing to give Vick his presumption of innocence.

Commentary, Ronvel Sharper. He is an American citizen, last I checked, and we don’t need to look any further than Duke lacrosse to see what can happen to a prosecutor when the media spotlight descends on a criminal case. Nope. My desire is to see Vick evolve as a human being and for his troubles to serve as yet another wake-up call for black athletes to reject the hip-hop/prison culture that glorifies much of the negative behavior and attitude that has eroded the once-dignified and positive reputation of African-American athletes. As much as I love dogs — and I really do have an affinity for them — this case primarily repulses me because I believe Vick got involved with breeding vicious pit bulls because rap-music culture made it the cool thing to do. Listen, I don’t want PETA supporters upset with me. Animal cruelty is intolerable.

But I’m wondering what could turn a human mind and heart so cold that a person would find pleasure in breeding dogs for cruel destruction in 2007. Seriously, Vick didn’t do it for the money. miserable. Vick was involved in pit bull breeding (and quite possibly dogfighting) because he enjoyed it. He’s a product of a culture that makes the “profession” acceptable and honorable. It’s the same culture that has turned the dope dealer into mayor of the neighborhood. It speaks to the grip the negative aspects of hip-hop culture have on young people.

Vick is a millionaire athlete who has spent most of his NFL career trying to maintain his street cred. Despite lifetime financial security, Mike Vick stayed on the “grind,” hustling for that paper with his Bad Newz Kennels. Idiot. Well, unless he plans on launching a rap career and releasing a solo “Dogfighting Was The Case,” I don’t see any of this ending well for Vick. If it doesn’t fit, you must acquit…even if it meant “one size fits all.” Apparently, that same Ditto, if it is the right “Ditto” (o rly?), could be the one responsible for frauding Michael Vick entirely…but investigators do not want to touch something that might give various viruses to anyone who touches it, especially STD’s. This is a cultural phenomenon that has swallowed a small percentage of African-American athletes, but a large enough percentage to significantly damage the overall perception of black, American-born athletes. As Dr.

Harry Edwards told me two weeks ago, it only takes a few key people to hijack an entire culture. N.W.A., the late-1980s rap group, hijacked hip-hop years ago, and calls to return it to something resembling decency and self-respect have fallen on Def Jam ear$. Allen Iverson and his sneaker/jersey sales hijacked the image of black professional athletes years ago, and out of fear of being labeled a racist or a sellout, few have even dared question the sanity of it … until now. Vick’s employer is in an impossible position. The right thing for the Falcons to do is support Vick through his legal proceedings. But how can the organization? Vick is a human distraction now.

Atlanta has a new coaching staff that will find it nearly impossible to operate smoothly in the environment/media circus Vick has created for the organization. Heck, even Al Sharpton and Russell Simmons joined in the castigation of Vick and dogfighting, penning a joint letter with PETA that was sent to NFL commissioner Roger Goodell and all of Vick’s corporate sponsors. True, the letter wasn’t all that harsh, but the fact that Sharpton would in any way publicly hold a black person responsible for any action is historic. And, if you have a scorebook at home, we now know that Russell Simmons is adamantly opposed to the killing and brutalization of dogs, but he is in favor of the glorification of killing black men in music. I’m just passing that along without any editorial comment. 2005: a guy, let’s call him doug, and a girl, let’s call her mary, have been ‘talking’ for some time. He shouldn’t be suspended or denied pay because the Falcons and the NFL have invested too much in Vick to treat him like Pacman Jones.

That’s right. I don’t believe in treating everyone the same. I believe in treating everyone fairly. Suspending Vick would be too prejudicial (legal term, not a race term) and inhibit his ability to receive a fair trial. If he’s convicted of a felony, the Falcons probably have provisions within his contract that would grant them the right to release him and go after a portion of his signing bonus if they so choose. Ray Lewis was at the scene of a double murder, failed initially to cooperate with police and eventually pled guilty to obstruction of justice charges. Ray used to be in love with his street cred, too.

It took double-murder charges to knock some sense into one of the game’s best linebackers. He evolved, and he’s certainly been an asset to the NFL ever since his evolution. Will the same thing happen to Michael Vick? I doubt it, but I certainly hope so. And, if you have a scorebook at home, we now know that Russell Simmons is adamantly opposed to the killing and brutalization of dogs, but he is in favor of the glorification of killing black men in music. And, if you have a scorebook at home, we now know that Russell Simmons is adamantly opposed to the killing and brutalization of dogs, but he is in favor of the glorification of killing black men in music. No matter what Vick did or did not to…

the above is a little disturbing to me. The media is constantly criticized for convicting someone upon accusation. This is another example. Posted by PAULSTORM (Member # 7261) on July 23, 2007, 08:28 AM: What is the word on the status of Vick playin for the Falcons this year? I think I heard O’Reilly say on his show that Vick will be playing this yr.